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Corrigendum.

Journal of immunotoxicology

[No authors listed]
PMID: 27278247
J Immunotoxicol. 2016 Mar;13(2):i. doi: 10.3109/1547691X.2016.1147889.

No abstract available.

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Corrigendum. J Immunotoxicol. 2016;13(2):idoi: 10.3109/1547691X.2016.1147889.
(2016). Corrigendum. Journal of immunotoxicology, 13(2), i. https://doi.org/10.3109/1547691X.2016.1147889
"Corrigendum." Journal of immunotoxicology vol. 13,2 (2016): i. doi: https://doi.org/10.3109/1547691X.2016.1147889
Corrigendum. J Immunotoxicol. 2016 Mar;13(2):i. doi: 10.3109/1547691X.2016.1147889. PMID: 27278247.
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Ultraviolet light and resistance to infectious diseases.

Journal of immunotoxicology

Sleijffers A, Garssen J, Vos JG, Loveren H.
PMID: 18958636
J Immunotoxicol. 2004 Jan;1(1):3-14. doi: 10.1080/15476910490438333.

Exposure to ultraviolet (UV) radiation, as in sunlight, can modulate immune responses in animals and humans. This immunomodulation can lead to positive health effects especially with respect to certain autoimmune diseases and allergies. However, UV-induced immunomodulation has also been...

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Sleijffers A, Garssen J, Vos JG, et al. Ultraviolet light and resistance to infectious diseases. J Immunotoxicol. 2004;1(1):3-14doi: 10.1080/15476910490438333.
Sleijffers, A., Garssen, J., Vos, J. G., & Loveren, H. (2004). Ultraviolet light and resistance to infectious diseases. Journal of immunotoxicology, 1(1), 3-14. https://doi.org/10.1080/15476910490438333
Sleijffers, Annemarie, et al. "Ultraviolet light and resistance to infectious diseases." Journal of immunotoxicology vol. 1,1 (2004): 3-14. doi: https://doi.org/10.1080/15476910490438333
Sleijffers A, Garssen J, Vos JG, Loveren H. Ultraviolet light and resistance to infectious diseases. J Immunotoxicol. 2004 Jan;1(1):3-14. doi: 10.1080/15476910490438333. PMID: 18958636.
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Immunologic effector mechanisms in animal models of occupational asthma.

Journal of immunotoxicology

Regal JF.
PMID: 18958638
J Immunotoxicol. 2004 Jan;1(1):25-37. doi: 10.1080/15476910490438351.

Occupational asthma is a form of immunotoxicity resulting from an exaggerated immune response to substances encountered in the workplace. Symptoms include reversible airway obstruction, airway hyperresponsiveness, airway remodeling, mucus production and cellular infiltration into the lung, particularly eosinophilia. The...

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Regal JF. Immunologic effector mechanisms in animal models of occupational asthma. J Immunotoxicol. 2004;1(1):25-37doi: 10.1080/15476910490438351.
Regal, J. F. (2004). Immunologic effector mechanisms in animal models of occupational asthma. Journal of immunotoxicology, 1(1), 25-37. https://doi.org/10.1080/15476910490438351
Regal, Jean F. "Immunologic effector mechanisms in animal models of occupational asthma." Journal of immunotoxicology vol. 1,1 (2004): 25-37. doi: https://doi.org/10.1080/15476910490438351
Regal JF. Immunologic effector mechanisms in animal models of occupational asthma. J Immunotoxicol. 2004 Jan;1(1):25-37. doi: 10.1080/15476910490438351. PMID: 18958638.
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Investigation of the involvement of macrophages and T cells in D-penicillamine-induced autoimmunity in the Brown Norway rat.

Journal of immunotoxicology

Masson MJ, Teranishi M, Shenton JM, Uetrecht JP.
PMID: 18958641
J Immunotoxicol. 2004 Apr;1(2):79-93. doi: 10.1080/15476910490496258.

The initiating events in drug-induced autoimmunity are poorly understood and difficult to study. We examined the role of macrophages and T-cells in the Brown Norway rat model of D-penicillamine-induced autoimmunity. When activated, macrophages can act as both antigen presenting...

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Masson MJ, Teranishi M, Shenton JM, et al. Investigation of the involvement of macrophages and T cells in D-penicillamine-induced autoimmunity in the Brown Norway rat. J Immunotoxicol. 2004;1(2):79-93doi: 10.1080/15476910490496258.
Masson, M. J., Teranishi, M., Shenton, J. M., & Uetrecht, J. P. (2004). Investigation of the involvement of macrophages and T cells in D-penicillamine-induced autoimmunity in the Brown Norway rat. Journal of immunotoxicology, 1(2), 79-93. https://doi.org/10.1080/15476910490496258
Masson, Mary Jane, et al. "Investigation of the involvement of macrophages and T cells in D-penicillamine-induced autoimmunity in the Brown Norway rat." Journal of immunotoxicology vol. 1,2 (2004): 79-93. doi: https://doi.org/10.1080/15476910490496258
Masson MJ, Teranishi M, Shenton JM, Uetrecht JP. Investigation of the involvement of macrophages and T cells in D-penicillamine-induced autoimmunity in the Brown Norway rat. J Immunotoxicol. 2004 Apr;1(2):79-93. doi: 10.1080/15476910490496258. PMID: 18958641.
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Trichloroethylene Does Not Accelerate Autoimmune Diabetes in NOD Mice.

Journal of immunotoxicology

Ravel G, Christ M, Perron-Lepage MF, Condevaux F, Descotes J.
PMID: 18958647
J Immunotoxicol. 2005 Jul 01;1(3):141-8. doi: 10.1080/15476910490916044.

Pre-existing or contributing risk factors, including genetic predisposition and environmental influences, are largely thought to play a crucial (though ill-elucidated) role in the development of autoimmunity. Trichloroethylene (TCE) is a widely used organic solvent, which has been suspected of...

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Ravel G, Christ M, Perron-Lepage MF, et al. Trichloroethylene Does Not Accelerate Autoimmune Diabetes in NOD Mice. J Immunotoxicol. 2005;1(3):141-8doi: 10.1080/15476910490916044.
Ravel, G., Christ, M., Perron-Lepage, M. F., Condevaux, F., & Descotes, J. (2005). Trichloroethylene Does Not Accelerate Autoimmune Diabetes in NOD Mice. Journal of immunotoxicology, 1(3), 141-8. https://doi.org/10.1080/15476910490916044
Ravel, Guillaume, et al. "Trichloroethylene Does Not Accelerate Autoimmune Diabetes in NOD Mice." Journal of immunotoxicology vol. 1,3 (2005): 141-8. doi: https://doi.org/10.1080/15476910490916044
Ravel G, Christ M, Perron-Lepage MF, Condevaux F, Descotes J. Trichloroethylene Does Not Accelerate Autoimmune Diabetes in NOD Mice. J Immunotoxicol. 2005 Jul 01;1(3):141-8. doi: 10.1080/15476910490916044. PMID: 18958647.
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Screening New Drugs for Immunotoxic Potential: I. Assessment of the Effects of Conventional Nonsteroidal Anti-Inflammatory Drugs and Selective COX-2 Inhibitors on In Vitro and In Vivo Phagocytic Activity.

Journal of immunotoxicology

Furst SM, Komocsar WJ, Khan KN, White KL, Peachee VL, Mennear JH.
PMID: 18958648
J Immunotoxicol. 2005 Jul 01;1(3):149-58. doi: 10.1080/15476910490916828.

Although both experimental and clinical literature contain reports suggestive of associations between enhanced susceptibility to soft tissue infections and nonsteroidal anti-inflammatory drug (NSAID) use, the immunotoxicological potential of this class of therapeutic agents has not been thoroughly investigated. In...

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Furst SM, Komocsar WJ, Khan KN, et al. Screening New Drugs for Immunotoxic Potential: I. Assessment of the Effects of Conventional Nonsteroidal Anti-Inflammatory Drugs and Selective COX-2 Inhibitors on In Vitro and In Vivo Phagocytic Activity. J Immunotoxicol. 2005;1(3):149-58doi: 10.1080/15476910490916828.
Furst, S. M., Komocsar, W. J., Khan, K. N., White, K. L., Peachee, V. L., & Mennear, J. H. (2005). Screening New Drugs for Immunotoxic Potential: I. Assessment of the Effects of Conventional Nonsteroidal Anti-Inflammatory Drugs and Selective COX-2 Inhibitors on In Vitro and In Vivo Phagocytic Activity. Journal of immunotoxicology, 1(3), 149-58. https://doi.org/10.1080/15476910490916828
Furst, Sylvia M, et al. "Screening New Drugs for Immunotoxic Potential: I. Assessment of the Effects of Conventional Nonsteroidal Anti-Inflammatory Drugs and Selective COX-2 Inhibitors on In Vitro and In Vivo Phagocytic Activity." Journal of immunotoxicology vol. 1,3 (2005): 149-58. doi: https://doi.org/10.1080/15476910490916828
Furst SM, Komocsar WJ, Khan KN, White KL, Peachee VL, Mennear JH. Screening New Drugs for Immunotoxic Potential: I. Assessment of the Effects of Conventional Nonsteroidal Anti-Inflammatory Drugs and Selective COX-2 Inhibitors on In Vitro and In Vivo Phagocytic Activity. J Immunotoxicol. 2005 Jul 01;1(3):149-58. doi: 10.1080/15476910490916828. PMID: 18958648.
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Immunotoxicity of gallium arsenide on antigen presentation: comparative study of intratracheal and intraperitoneal exposure routes.

Journal of immunotoxicology

Hartmann CB, Harrison MT, McCoy KL.
PMID: 18958654
J Immunotoxicol. 2005 Jan 01;2(1):1-9. doi: 10.1080/15476910590930083.

Gallium arsenide (GaAs) is a semiconductor utilized in electronics and computer industries. GaAs exposure of animals causes local inflammation and systemic immune suppression. Mice were administered 2 to 200 mg/kg GaAs. On day 5, intratracheal instillation increased lung weights...

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Hartmann CB, Harrison MT, McCoy KL. Immunotoxicity of gallium arsenide on antigen presentation: comparative study of intratracheal and intraperitoneal exposure routes. J Immunotoxicol. 2005;2(1):1-9doi: 10.1080/15476910590930083.
Hartmann, C. B., Harrison, M. T., & McCoy, K. L. (2005). Immunotoxicity of gallium arsenide on antigen presentation: comparative study of intratracheal and intraperitoneal exposure routes. Journal of immunotoxicology, 2(1), 1-9. https://doi.org/10.1080/15476910590930083
Hartmann, Constance B, et al. "Immunotoxicity of gallium arsenide on antigen presentation: comparative study of intratracheal and intraperitoneal exposure routes." Journal of immunotoxicology vol. 2,1 (2005): 1-9. doi: https://doi.org/10.1080/15476910590930083
Hartmann CB, Harrison MT, McCoy KL. Immunotoxicity of gallium arsenide on antigen presentation: comparative study of intratracheal and intraperitoneal exposure routes. J Immunotoxicol. 2005 Jan 01;2(1):1-9. doi: 10.1080/15476910590930083. PMID: 18958654.
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Developmental Immunotoxicology of Di-(2-Ethylhexyl)phthalate (DEHP): Age-Based Assessment in the Female Rat.

Journal of immunotoxicology

Piepenbrink MS, Hussain I, Marsh JA, Dietert RR.
PMID: 18958656
J Immunotoxicol. 2005 Jan 01;2(1):21-31. doi: 10.1080/15363750490429435.

Di-(2-ethylhexyl)phthalate (DEHP) helps provide flexibility to plastic products including food containers and medical IV devices. Age-based sensitivity to DEHP-induced immunotoxicity was compared in female CD strain rats following administration for 16 consecutive days in utero vs. adult exposure. Pregnant...

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Piepenbrink MS, Hussain I, Marsh JA, et al. Developmental Immunotoxicology of Di-(2-Ethylhexyl)phthalate (DEHP): Age-Based Assessment in the Female Rat. J Immunotoxicol. 2005;2(1):21-31doi: 10.1080/15363750490429435.
Piepenbrink, M. S., Hussain, I., Marsh, J. A., & Dietert, R. R. (2005). Developmental Immunotoxicology of Di-(2-Ethylhexyl)phthalate (DEHP): Age-Based Assessment in the Female Rat. Journal of immunotoxicology, 2(1), 21-31. https://doi.org/10.1080/15363750490429435
Piepenbrink, Michael S, et al. "Developmental Immunotoxicology of Di-(2-Ethylhexyl)phthalate (DEHP): Age-Based Assessment in the Female Rat." Journal of immunotoxicology vol. 2,1 (2005): 21-31. doi: https://doi.org/10.1080/15363750490429435
Piepenbrink MS, Hussain I, Marsh JA, Dietert RR. Developmental Immunotoxicology of Di-(2-Ethylhexyl)phthalate (DEHP): Age-Based Assessment in the Female Rat. J Immunotoxicol. 2005 Jan 01;2(1):21-31. doi: 10.1080/15363750490429435. PMID: 18958656.
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Drug-induced type 1 and type 2 immune responses are characterized by distinct profiles of cell kinetics, cytokine production, and expression of co-stimulatory molecules in the popliteal lymph node assay.

Journal of immunotoxicology

Nierkens S, Aalbers M, Bleumink R, Boon L, Pieters R.
PMID: 18958667
J Immunotoxicol. 2005 Jul 01;2(3):141-50. doi: 10.1080/15476910500221067.

Some drugs have the undesired side effect of systemically stimulating the immune system, which may eventually lead to the development of drug-induced allergy or autoimmunity. Unfortunately, validated predictive screening tools to assess the immune stimulatory potential of compounds are...

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Nierkens S, Aalbers M, Bleumink R, et al. Drug-induced type 1 and type 2 immune responses are characterized by distinct profiles of cell kinetics, cytokine production, and expression of co-stimulatory molecules in the popliteal lymph node assay. J Immunotoxicol. 2005;2(3):141-50doi: 10.1080/15476910500221067.
Nierkens, S., Aalbers, M., Bleumink, R., Boon, L., & Pieters, R. (2005). Drug-induced type 1 and type 2 immune responses are characterized by distinct profiles of cell kinetics, cytokine production, and expression of co-stimulatory molecules in the popliteal lymph node assay. Journal of immunotoxicology, 2(3), 141-50. https://doi.org/10.1080/15476910500221067
Nierkens, Stefan, et al. "Drug-induced type 1 and type 2 immune responses are characterized by distinct profiles of cell kinetics, cytokine production, and expression of co-stimulatory molecules in the popliteal lymph node assay." Journal of immunotoxicology vol. 2,3 (2005): 141-50. doi: https://doi.org/10.1080/15476910500221067
Nierkens S, Aalbers M, Bleumink R, Boon L, Pieters R. Drug-induced type 1 and type 2 immune responses are characterized by distinct profiles of cell kinetics, cytokine production, and expression of co-stimulatory molecules in the popliteal lymph node assay. J Immunotoxicol. 2005 Jul 01;2(3):141-50. doi: 10.1080/15476910500221067. PMID: 18958667.
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Preclinical immunogenicity testing for recombinant therapeutic proteins.

Journal of immunotoxicology

Smith HW, Wierda D.
PMID: 18958675
J Immunotoxicol. 2005 Oct 01;2(4):203-10. doi: 10.1080/15476910500362911.

The assessment of the immunogenicity of recombinant therapeutic proteins (RTPs) has received more attention in the past 4 years than in the previous 20 years. The induction of clinically adverse events in RTP-treated patients and the subsequent measurement of...

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Smith HW, Wierda D. Preclinical immunogenicity testing for recombinant therapeutic proteins. J Immunotoxicol. 2005;2(4):203-10doi: 10.1080/15476910500362911.
Smith, H. W., & Wierda, D. (2005). Preclinical immunogenicity testing for recombinant therapeutic proteins. Journal of immunotoxicology, 2(4), 203-10. https://doi.org/10.1080/15476910500362911
Smith, Holly W, and Wierda, Daniel. "Preclinical immunogenicity testing for recombinant therapeutic proteins." Journal of immunotoxicology vol. 2,4 (2005): 203-10. doi: https://doi.org/10.1080/15476910500362911
Smith HW, Wierda D. Preclinical immunogenicity testing for recombinant therapeutic proteins. J Immunotoxicol. 2005 Oct 01;2(4):203-10. doi: 10.1080/15476910500362911. PMID: 18958675.
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Pulmonary immunotoxic potentials of metals are governed by select physicochemical properties: chromium agents.

Journal of immunotoxicology

Cohen MD, Prophete C, Sisco M, Chen LC, Zelikoff JT, Smee JJ, Holder AA, Crans DC.
PMID: 18958687
J Immunotoxicol. 2006 Jul 01;3(2):69-81. doi: 10.1080/15476910600718434.

Increasing the understanding of how metal ions/complexes react in situ will allow for the improved specificity and controlled toxicity of novel synthetic metallocompounds that will be used as inhaled diagnostics or therapeutics. Our previous work showed that inhalation of...

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Cohen MD, Prophete C, Sisco M, et al. Pulmonary immunotoxic potentials of metals are governed by select physicochemical properties: chromium agents. J Immunotoxicol. 2006;3(2):69-81doi: 10.1080/15476910600718434.
Cohen, M. D., Prophete, C., Sisco, M., Chen, L. C., Zelikoff, J. T., Smee, J. J., Holder, A. A., & Crans, D. C. (2006). Pulmonary immunotoxic potentials of metals are governed by select physicochemical properties: chromium agents. Journal of immunotoxicology, 3(2), 69-81. https://doi.org/10.1080/15476910600718434
Cohen, Mitchell D, et al. "Pulmonary immunotoxic potentials of metals are governed by select physicochemical properties: chromium agents." Journal of immunotoxicology vol. 3,2 (2006): 69-81. doi: https://doi.org/10.1080/15476910600718434
Cohen MD, Prophete C, Sisco M, Chen LC, Zelikoff JT, Smee JJ, Holder AA, Crans DC. Pulmonary immunotoxic potentials of metals are governed by select physicochemical properties: chromium agents. J Immunotoxicol. 2006 Jul 01;3(2):69-81. doi: 10.1080/15476910600718434. PMID: 18958687.
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Strategies and assays for the assessment of unwanted immunogenicity.

Journal of immunotoxicology

Wadhwa M, Thorpe R.
PMID: 18958691
J Immunotoxicol. 2006 Sep 01;3(3):115-21. doi: 10.1080/15476910600845534.

The assessment of unwanted immunogenicity associated with biological products continues to be a major issue for the biotechnology industry. Monitoring of unwanted immunogenicity during the development of the product from pre-clinical stage through clinical trials is now a regulatory...

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Wadhwa M, Thorpe R. Strategies and assays for the assessment of unwanted immunogenicity. J Immunotoxicol. 2006;3(3):115-21doi: 10.1080/15476910600845534.
Wadhwa, M., & Thorpe, R. (2006). Strategies and assays for the assessment of unwanted immunogenicity. Journal of immunotoxicology, 3(3), 115-21. https://doi.org/10.1080/15476910600845534
Wadhwa, Meenu, and Thorpe, Robin. "Strategies and assays for the assessment of unwanted immunogenicity." Journal of immunotoxicology vol. 3,3 (2006): 115-21. doi: https://doi.org/10.1080/15476910600845534
Wadhwa M, Thorpe R. Strategies and assays for the assessment of unwanted immunogenicity. J Immunotoxicol. 2006 Sep 01;3(3):115-21. doi: 10.1080/15476910600845534. PMID: 18958691.
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Showing 1 to 12 of 128 entries